Cargando…
Hyperglycemia induced damage to mitochondrial respiration in renal mesangial and tubular cells: Implications for diabetic nephropathy
Damage to renal tubular and mesangial cells is central to the development of diabetic nephropathy (DN), a complication of diabetes which can lead to renal failure. Mitochondria are the site of cellular respiration and produce energy in the form of ATP via oxidative phosphorylation, and mitochondrial...
Autores principales: | Czajka, Anna, Malik, Afshan N. |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Elsevier
2016
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5053113/ https://www.ncbi.nlm.nih.gov/pubmed/27710853 http://dx.doi.org/10.1016/j.redox.2016.09.007 |
Ejemplares similares
-
Altered Mitochondrial Function, Mitochondrial DNA and Reduced Metabolic Flexibility in Patients With Diabetic Nephropathy
por: Czajka, Anna, et al.
Publicado: (2015) -
Changes in the gene expression programs of renal mesangial cells during diabetic nephropathy
por: Brunskill, Eric W, et al.
Publicado: (2012) -
Hyperglycemia Causes Renal Cell Damage via CCN2-Induced Activation of the TrkA Receptor: Implications for Diabetic Nephropathy
por: Fragiadaki, Maria, et al.
Publicado: (2012) -
Pathophysiology of mesangial expansion in diabetic nephropathy: mesangial structure, glomerular biomechanics, and biochemical signaling and regulation
por: Thomas, Haryana Y., et al.
Publicado: (2022) -
Effect of Cysteine on Methylglyoxal-Induced Renal Damage in Mesangial Cells
por: Lee, Jae Hyuk, et al.
Publicado: (2020)