Cargando…
Prolonged feeding with guanidinoacetate, a methyl group consumer, exacerbates ethanol-induced liver injury
AIM: To investigate the hypothesis that exposure to guanidinoacetate (GAA, a potent methyl-group consumer) either alone or combined with ethanol intake for a prolonged period of time would cause more advanced liver pathology thus identifying methylation defects as the initiator and stimulator for pr...
Autores principales: | Osna, Natalia A, Feng, Dan, Ganesan, Murali, Maillacheruvu, Priya F, Orlicky, David J, French, Samuel W, Tuma, Dean J, Kharbanda, Kusum K |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Baishideng Publishing Group Inc
2016
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5064031/ https://www.ncbi.nlm.nih.gov/pubmed/27784962 http://dx.doi.org/10.3748/wjg.v22.i38.8497 |
Ejemplares similares
-
Obeticholic acid attenuates human immunodeficiency virus/alcohol metabolism-induced pro-fibrotic activation in liver cells
por: New-Aaron, Moses, et al.
Publicado: (2020) -
Hepatitis C Virus-Infected Apoptotic Hepatocytes Program Macrophages and Hepatic Stellate Cells for Liver Inflammation and Fibrosis Development: Role of Ethanol as a Second Hit
por: Ganesan, Murali, et al.
Publicado: (2018) -
Hepatitis C, Innate Immunity and Alcohol: Friends or Foes?
por: Osna, Natalia A., et al.
Publicado: (2015) -
Demethylase JMJD6 as a New Regulator of Interferon Signaling: Effects of HCV and Ethanol Metabolism
por: Ganesan, Murali, et al.
Publicado: (2017) -
Human immunodeficiency virus and hepatotropic viruses co-morbidities as the inducers of liver injury progression
por: Ganesan, Murali, et al.
Publicado: (2019)