Tumor necrosis factor-mediated inhibition of interleukin-18 in the brain: a clinical and experimental study in head-injured patients and in a murine model of closed head injury.

Tumor necrosis factor (TNF) and interleukin-(IL)-18 are important mediators of neuroinflammation after closed head injury (CHI). Both mediators have been previously found to be significantly elevated in the intracranial compartment after brain injury, both in patients as well as in experimental mode...

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Detalles Bibliográficos
Autores principales: Schmidt, Oliver I, Morganti-Kossmann, Maria Cristina, Heyde, Christoph E, Perez, Daniel, Yatsiv, Ido, Shohami, Esther, Ertel, Wolfgang, Stahel, Philip F
Formato: Texto
Lenguaje:English
Publicado: BioMed Central 2004
Materias:
Short Report
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC506787/
https://www.ncbi.nlm.nih.gov/pubmed/15285802
http://dx.doi.org/10.1186/1742-2094-1-13

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC506787/
https://www.ncbi.nlm.nih.gov/pubmed/15285802
http://dx.doi.org/10.1186/1742-2094-1-13

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