Cargando…
Complex Breakpoints and Template Switching Associated with Non-canonical Termination of Homologous Recombination in Mammalian Cells
A proportion of homologous recombination (HR) events in mammalian cells resolve by “long tract” gene conversion, reflecting copying of several kilobases from the donor sister chromatid prior to termination. Cells lacking the major hereditary breast/ovarian cancer predisposition genes, BRCA1 or BRCA2...
Autores principales: | Hartlerode, Andrea J., Willis, Nicholas A., Rajendran, Anbazhagan, Manis, John P., Scully, Ralph |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Public Library of Science
2016
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5104497/ https://www.ncbi.nlm.nih.gov/pubmed/27832076 http://dx.doi.org/10.1371/journal.pgen.1006410 |
Ejemplares similares
-
Correction: Complex Breakpoints and Template Switching Associated with Non-canonical Termination of Homologous Recombination in Mammalian Cells
por: Hartlerode, Andrea J., et al.
Publicado: (2016) -
Cell Cycle-Dependent Induction of Homologous Recombination by a Tightly Regulated I-SceI Fusion Protein
por: Hartlerode, Andrea, et al.
Publicado: (2011) -
Rad51 recruitment and exclusion of non-homologous end joining during homologous recombination at a Tus/Ter mammalian replication fork barrier
por: Willis, Nicholas A., et al.
Publicado: (2018) -
BRCA1 controls homologous recombination at Tus/Ter-stalled mammalian replication forks
por: Willis, Nicholas A., et al.
Publicado: (2014) -
Impact of Histone H4 Lysine 20 Methylation on 53BP1 Responses to Chromosomal Double Strand Breaks
por: Hartlerode, Andrea J., et al.
Publicado: (2012)