Cargando…
Lack of an apparent role for endothelin‐1 in the prolonged reduction in renal perfusion following severe unilateral ischemia‐reperfusion injury in the mouse
Therapeutic approaches to block the progression from acute kidney injury to chronic kidney disease are currently lacking. Endothelin‐1 (ET‐1) is a powerful vasoconstrictor, induced by hypoxia, and previously implicated in renal ischemia‐reperfusion (IR) injury. This study tested the hypothesis that...
Autor principal: | Boesen, Erika I. |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
John Wiley and Sons Inc.
2016
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5112503/ https://www.ncbi.nlm.nih.gov/pubmed/27905299 http://dx.doi.org/10.14814/phy2.13027 |
Ejemplares similares
-
“Point of no return” in unilateral renal ischemia reperfusion injury in mice
por: Holderied, Alexander, et al.
Publicado: (2020) -
PARP Inhibition Attenuates Histopathological Lesion in Ischemia/Reperfusion Renal Mouse Model after Cold Prolonged Ischemia
por: del Moral, Raimundo M. G., et al.
Publicado: (2013) -
ET(A) receptor activation contributes to T cell accumulation in the kidney following ischemia‐reperfusion injury
por: Boesen, Erika I.
Publicado: (2018) -
The creation of unilateral intermittent and unintermittent renal ischemia-reperfusion models in rats
por: Soytas, Mustafa, et al.
Publicado: (2021) -
Effects of Endothelin-1 and nitric oxide levels on myocardial ischemia-reperfusion injury
por: Ai, Wenwei, et al.
Publicado: (2022)