Pancreatic α-cell hyperplasia and hyperglucagonemia due to a glucagon receptor splice mutation

Glucagon stimulates hepatic glucose production by activating specific glucagon receptors in the liver, which in turn increase hepatic glycogenolysis as well as gluconeogenesis and ureagenesis from amino acids. Conversely, glucagon secretion is regulated by concentrations of glucose and amino acids....

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Detalles Bibliográficos
Autores principales: Larger, Etienne, Wewer Albrechtsen, Nicolai J, Hansen, Lars H, Gelling, Richard W, Capeau, Jacqueline, Deacon, Carolyn F, Madsen, Ole D, Yakushiji, Fumiatsu, De Meyts, Pierre, Holst, Jens J, Nishimura, Erica
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Bioscientifica Ltd 2016
Materias:
Insight into Disease Pathogenesis or Mechanism of Therapy
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5118975/
https://www.ncbi.nlm.nih.gov/pubmed/27933176
http://dx.doi.org/10.1530/EDM-16-0081

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