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Correction: HMGB1-RAGE Axis Makes No Contribution to Cardiac Remodeling Induced by Pressure-Overload

Detalles Bibliográficos
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Public Library of Science 2016
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5135125/
https://www.ncbi.nlm.nih.gov/pubmed/27911945
http://dx.doi.org/10.1371/journal.pone.0167872
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spelling pubmed-51351252016-12-21 Correction: HMGB1-RAGE Axis Makes No Contribution to Cardiac Remodeling Induced by Pressure-Overload PLoS One Correction Public Library of Science 2016-12-02 /pmc/articles/PMC5135125/ /pubmed/27911945 http://dx.doi.org/10.1371/journal.pone.0167872 Text en © 2016 The PLOS ONE Staff http://creativecommons.org/licenses/by/4.0/ This is an open access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/) , which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.
spellingShingle Correction
Correction: HMGB1-RAGE Axis Makes No Contribution to Cardiac Remodeling Induced by Pressure-Overload
title Correction: HMGB1-RAGE Axis Makes No Contribution to Cardiac Remodeling Induced by Pressure-Overload
title_full Correction: HMGB1-RAGE Axis Makes No Contribution to Cardiac Remodeling Induced by Pressure-Overload
title_fullStr Correction: HMGB1-RAGE Axis Makes No Contribution to Cardiac Remodeling Induced by Pressure-Overload
title_full_unstemmed Correction: HMGB1-RAGE Axis Makes No Contribution to Cardiac Remodeling Induced by Pressure-Overload
title_short Correction: HMGB1-RAGE Axis Makes No Contribution to Cardiac Remodeling Induced by Pressure-Overload
title_sort correction: hmgb1-rage axis makes no contribution to cardiac remodeling induced by pressure-overload
topic Correction
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5135125/
https://www.ncbi.nlm.nih.gov/pubmed/27911945
http://dx.doi.org/10.1371/journal.pone.0167872
work_keys_str_mv AT correctionhmgb1rageaxismakesnocontributiontocardiacremodelinginducedbypressureoverload