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Cigarette smoke extract profoundly suppresses TNFα-mediated proinflammatory gene expression through upregulation of ATF3 in human coronary artery endothelial cells

Endothelial dysfunction caused by the combined action of disturbed flow, inflammatory mediators and oxidants derived from cigarette smoke is known to promote coronary atherosclerosis and increase the likelihood of myocardial infarctions and strokes. Conversely, laminar flow protects against endothel...

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Detalles Bibliográficos
Autores principales:	Teasdale, Jack E., Hazell, Georgina G. J., Peachey, Alasdair M. G., Sala-Newby, Graciela B., Hindmarch, Charles C. T., McKay, Tristan R., Bond, Mark, Newby, Andrew C., White, Stephen J.
Formato:	Online Artículo Texto
Lenguaje:	English
Publicado:	Nature Publishing Group 2017
Materias:	Article
Acceso en línea:	https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5216376/ https://www.ncbi.nlm.nih.gov/pubmed/28059114 http://dx.doi.org/10.1038/srep39945

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