Response of narrow QRS tachycardia to premature atrial extra. What is the mechanism?

A 60-year-old woman was referred for radiofrequency catheter ablation of narrow QRS tachycardia that was terminated with intravenous adenosine. Twelve-lead ECG showed no baseline preexcitation. Echocardiogram was essentially normal. The electrophysiological study showed a normal AH interval of 114 m...

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Detalles Bibliográficos
Autores principales: Mohanan Nair, Krishna Kumar, Namboodiri, Narayanan, Banavalikar, Bharatraj, Abhilash, Sreevilasam Pushpangadhan, Thajudeen, Anees, Valaparambil, Ajitkumar
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Elsevier 2016
Materias:
Device/Arrhythmia round
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5219836/
https://www.ncbi.nlm.nih.gov/pubmed/28401870
http://dx.doi.org/10.1016/j.ipej.2016.11.010
Descripción
Sumario:A 60-year-old woman was referred for radiofrequency catheter ablation of narrow QRS tachycardia that was terminated with intravenous adenosine. Twelve-lead ECG showed no baseline preexcitation. Echocardiogram was essentially normal. The electrophysiological study showed a normal AH interval of 114 ms and HV interval of 48 ms during sinus rhythm. Anterograde study demonstrated no dual AV nodal physiology. Atrial pacing protocols easily and reproducibly induced narrow QRS tachycardia (Fig. 1A & 1B). Premature atrial extra (PAE) stimuli were delivered during the tachycardia (Fig. 2). What is the mechanism of the tachycardia?

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