Cargando…
TrkB-containing exosomes promote the transfer of glioblastoma aggressiveness to YKL-40-inactivated glioblastoma cells
The neurotrophin receptors are known to promote growth and proliferation of glioblastoma cells. Their functions in spreading glioblastoma cell aggressiveness to the microenvironment through exosome release from glioblastoma cells are unknown. Considering previous reports demonstrating that YKL-40 ex...
Autores principales: | Pinet, Sandra, Bessette, Barbara, Vedrenne, Nicolas, Lacroix, Aurélie, Richard, Laurence, Jauberteau, Marie-Odile, Battu, Serge, Lalloué, Fabrice |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Impact Journals LLC
2016
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5226587/ https://www.ncbi.nlm.nih.gov/pubmed/27385098 http://dx.doi.org/10.18632/oncotarget.10387 |
Ejemplares similares
-
KLRC3, a Natural Killer receptor gene, is a key factor involved in glioblastoma tumourigenesis and aggressiveness
por: Cheray, Mathilde, et al.
Publicado: (2016) -
SCO-spondin oligopeptide inhibits angiogenesis in glioblastoma
por: Bibes, Romain, et al.
Publicado: (2017) -
Autophagy inhibition reinforces stemness together with exit from dormancy of polydisperse glioblastoma stem cells
por: Brunel, Aude, et al.
Publicado: (2021) -
Dual inhibition of BDNF/TrkB and autophagy: a promising therapeutic approach for colorectal cancer
por: Mazouffre, Clément, et al.
Publicado: (2017) -
Fine-Tuning Roles of Endogenous Brain-Derived Neurotrophic Factor, TrkB and Sortilin in Colorectal Cancer Cell Survival
por: Akil, Hussein, et al.
Publicado: (2011)