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Ameliorative effects of Compound K and ginsenoside Rh1 on non-alcoholic fatty liver disease in rats
Non-alcoholic fatty liver disease (NAFLD) is a common liver disease, which has no standard treatment available. Panax notoginseng saponines (PNS) have recently been reported to protect liver against hepatocyte injury induced by ethanol or high fat diet (HFD) in rats. Compound K and ginsenoside Rh1 a...
Autores principales: | , , , , , , , , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Nature Publishing Group
2017
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5247696/ https://www.ncbi.nlm.nih.gov/pubmed/28106137 http://dx.doi.org/10.1038/srep41144 |
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author | Chen, Xu-Jia Liu, Wen-Jing Wen, Meng-Liang Liang, Hong Wu, Shao-Mei Zhu, Yun-Zhen Zhao, Jiang-Yuan Dong, Xiang-Qian Li, Ming-Gang Bian, Li Zou, Cheng-Gang Ma, Lan-Qing |
author_facet | Chen, Xu-Jia Liu, Wen-Jing Wen, Meng-Liang Liang, Hong Wu, Shao-Mei Zhu, Yun-Zhen Zhao, Jiang-Yuan Dong, Xiang-Qian Li, Ming-Gang Bian, Li Zou, Cheng-Gang Ma, Lan-Qing |
author_sort | Chen, Xu-Jia |
collection | PubMed |
description | Non-alcoholic fatty liver disease (NAFLD) is a common liver disease, which has no standard treatment available. Panax notoginseng saponines (PNS) have recently been reported to protect liver against hepatocyte injury induced by ethanol or high fat diet (HFD) in rats. Compound K and ginsenoside Rh1 are the main metabolites of PNS. In this study, we evaluated the effects of CK and Rh1 on NAFLD. Rats fed HFD showed significant elevations in liver function markers, lipids, glucose tolerance, and insulin resistance. Treatment with CK or Rh1 either alone or in combination dramatically ameliorated the liver function impairment induced by HFD. Histologically, CK and Rh1 significantly reversed HFD-induced hepatocyte injury and liver fibrosis. In vitro experiments demonstrated that treatment with CK or Rh1 alone or in combination markedly induced cell apoptosis, and inhibited cell proliferation and activation in HSC-T6 cells. Additionally, CK and Rh1, either alone or in combination, also repressed the expression of fibrotic factors TIMP-1, PC-I, and PC-III. Taken together, our results demonstrate that CK and Rh1 have positive effects on NAFLD via the anti-fibrotic and hepatoprotective activity. |
format | Online Article Text |
id | pubmed-5247696 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2017 |
publisher | Nature Publishing Group |
record_format | MEDLINE/PubMed |
spelling | pubmed-52476962017-01-23 Ameliorative effects of Compound K and ginsenoside Rh1 on non-alcoholic fatty liver disease in rats Chen, Xu-Jia Liu, Wen-Jing Wen, Meng-Liang Liang, Hong Wu, Shao-Mei Zhu, Yun-Zhen Zhao, Jiang-Yuan Dong, Xiang-Qian Li, Ming-Gang Bian, Li Zou, Cheng-Gang Ma, Lan-Qing Sci Rep Article Non-alcoholic fatty liver disease (NAFLD) is a common liver disease, which has no standard treatment available. Panax notoginseng saponines (PNS) have recently been reported to protect liver against hepatocyte injury induced by ethanol or high fat diet (HFD) in rats. Compound K and ginsenoside Rh1 are the main metabolites of PNS. In this study, we evaluated the effects of CK and Rh1 on NAFLD. Rats fed HFD showed significant elevations in liver function markers, lipids, glucose tolerance, and insulin resistance. Treatment with CK or Rh1 either alone or in combination dramatically ameliorated the liver function impairment induced by HFD. Histologically, CK and Rh1 significantly reversed HFD-induced hepatocyte injury and liver fibrosis. In vitro experiments demonstrated that treatment with CK or Rh1 alone or in combination markedly induced cell apoptosis, and inhibited cell proliferation and activation in HSC-T6 cells. Additionally, CK and Rh1, either alone or in combination, also repressed the expression of fibrotic factors TIMP-1, PC-I, and PC-III. Taken together, our results demonstrate that CK and Rh1 have positive effects on NAFLD via the anti-fibrotic and hepatoprotective activity. Nature Publishing Group 2017-01-20 /pmc/articles/PMC5247696/ /pubmed/28106137 http://dx.doi.org/10.1038/srep41144 Text en Copyright © 2017, The Author(s) http://creativecommons.org/licenses/by/4.0/ This work is licensed under a Creative Commons Attribution 4.0 International License. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in the credit line; if the material is not included under the Creative Commons license, users will need to obtain permission from the license holder to reproduce the material. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/ |
spellingShingle | Article Chen, Xu-Jia Liu, Wen-Jing Wen, Meng-Liang Liang, Hong Wu, Shao-Mei Zhu, Yun-Zhen Zhao, Jiang-Yuan Dong, Xiang-Qian Li, Ming-Gang Bian, Li Zou, Cheng-Gang Ma, Lan-Qing Ameliorative effects of Compound K and ginsenoside Rh1 on non-alcoholic fatty liver disease in rats |
title | Ameliorative effects of Compound K and ginsenoside Rh1 on non-alcoholic fatty liver disease in rats |
title_full | Ameliorative effects of Compound K and ginsenoside Rh1 on non-alcoholic fatty liver disease in rats |
title_fullStr | Ameliorative effects of Compound K and ginsenoside Rh1 on non-alcoholic fatty liver disease in rats |
title_full_unstemmed | Ameliorative effects of Compound K and ginsenoside Rh1 on non-alcoholic fatty liver disease in rats |
title_short | Ameliorative effects of Compound K and ginsenoside Rh1 on non-alcoholic fatty liver disease in rats |
title_sort | ameliorative effects of compound k and ginsenoside rh1 on non-alcoholic fatty liver disease in rats |
topic | Article |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5247696/ https://www.ncbi.nlm.nih.gov/pubmed/28106137 http://dx.doi.org/10.1038/srep41144 |
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