Rab32 connects ER stress to mitochondrial defects in multiple sclerosis

BACKGROUND: Endoplasmic reticulum (ER) stress is a hallmark of neurodegenerative diseases such as multiple sclerosis (MS). However, this physiological mechanism has multiple manifestations that range from impaired clearance of unfolded proteins to altered mitochondrial dynamics and apoptosis. While...

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Detalles Bibliográficos
Autores principales: Haile, Yohannes, Deng, Xiaodan, Ortiz-Sandoval, Carolina, Tahbaz, Nasser, Janowicz, Aleksandra, Lu, Jian-Qiang, Kerr, Bradley J., Gutowski, Nicholas J., Holley, Janet E., Eggleton, Paul, Giuliani, Fabrizio, Simmen, Thomas
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BioMed Central 2017
Materias:
Research
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5260063/
https://www.ncbi.nlm.nih.gov/pubmed/28115010
http://dx.doi.org/10.1186/s12974-016-0788-z

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