TRIP13-deficient tubular epithelial cells are susceptible to apoptosis following acute kidney injury

Damage to renal tubular epithelial cells by genetic, environmental, or biological insults can initiate complex signaling mechanisms that promote kidney repair and functional recovery. In this study, we demonstrated that thyroid receptor interacting protein 13 (TRIP13) is a critical modulator of tubu...

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Detalles Bibliográficos
Autores principales: Pressly, Jeffrey D., Hama, Taketsugu, Brien, Shannon O’, Regner, Kevin R., Park, Frank
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Nature Publishing Group 2017
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5335694/
https://www.ncbi.nlm.nih.gov/pubmed/28256593
http://dx.doi.org/10.1038/srep43196

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5335694/
https://www.ncbi.nlm.nih.gov/pubmed/28256593
http://dx.doi.org/10.1038/srep43196

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