Cargando…
IL-22, GM-CSF and IL-17 in peripheral CD4(+) T cell subpopulations during multiple sclerosis relapses and remission. Impact of corticosteroid therapy
Multiple sclerosis (MS) is thought to be a Th17-mediated dysimmune disease of the central nervous system. However, recent publications have questioned the pathogenicity of IL-17 per se and rather suggest the implication of other Th17-related inflammatory mediators. Therefore, we studied the expressi...
Autores principales: | Muls, Nathalie, Nasr, Zakia, Dang, Hong Anh, Sindic, Christian, van Pesch, Vincent |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Public Library of Science
2017
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5354390/ https://www.ncbi.nlm.nih.gov/pubmed/28301515 http://dx.doi.org/10.1371/journal.pone.0173780 |
Ejemplares similares
-
Fingolimod Increases CD39-Expressing Regulatory T Cells in Multiple Sclerosis Patients
por: Muls, Nathalie, et al.
Publicado: (2014) -
IL-27, but not IL-35, inhibits neuroinflammation through modulating GM-CSF expression
por: Casella, Giacomo, et al.
Publicado: (2017) -
GM-CSF Signalling Boosts Dramatically IL-1Production
por: Khameneh, Hanif Javanmard, et al.
Publicado: (2011) -
Innate Immune Modulation by GM-CSF and IL-3 in Health and Disease
por: Borriello, Francesco, et al.
Publicado: (2019) -
Irradiated Bladder Cancer Cells Expressing both GM-CSF and IL-21 versus Either GM-CSF or IL-21 Alone as Tumor Vaccine in a Mouse Xenograft Model
por: Peng, Junming, et al.
Publicado: (2019)