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Multiple acquired portosystemic shunts in a cat secondary to chronic diaphragmatic rupture

CASE SUMMARY: A cat with a chronic diaphragmatic rupture presented with neurological signs, including twitching and focal seizures. Blood ammonia level was markedly elevated and therefore neurological signs were thought to be related to hepatic encephalopathy. Exploratory laparotomy revealed that th...

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Autores principales: Barfield, Dominic M, Gibson, Andrew D, Lipscomb, Vicky J
Formato: Online Artículo Texto
Lenguaje:English
Publicado: SAGE Publications 2015
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5362922/
https://www.ncbi.nlm.nih.gov/pubmed/28491353
http://dx.doi.org/10.1177/2055116915585020
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author Barfield, Dominic M
Gibson, Andrew D
Lipscomb, Vicky J
author_facet Barfield, Dominic M
Gibson, Andrew D
Lipscomb, Vicky J
author_sort Barfield, Dominic M
collection PubMed
description CASE SUMMARY: A cat with a chronic diaphragmatic rupture presented with neurological signs, including twitching and focal seizures. Blood ammonia level was markedly elevated and therefore neurological signs were thought to be related to hepatic encephalopathy. Exploratory laparotomy revealed that the left lateral and medial liver lobes were herniated into the thorax and multiple acquired portosystemic shunts (MAPSS) were present. The hernia was reduced and the diaphragm repaired. Neurological signs gradually resolved following surgery and 1 year postoperatively the cat was clinically normal, was not on any medication and had no evidence of hepatic dysfunction. RELEVANCE AND NOVEL INFORMATION: This is the first report of a chronic diaphragmatic rupture leading to MAPSS in a cat.
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spelling pubmed-53629222017-05-10 Multiple acquired portosystemic shunts in a cat secondary to chronic diaphragmatic rupture Barfield, Dominic M Gibson, Andrew D Lipscomb, Vicky J JFMS Open Rep Case Report CASE SUMMARY: A cat with a chronic diaphragmatic rupture presented with neurological signs, including twitching and focal seizures. Blood ammonia level was markedly elevated and therefore neurological signs were thought to be related to hepatic encephalopathy. Exploratory laparotomy revealed that the left lateral and medial liver lobes were herniated into the thorax and multiple acquired portosystemic shunts (MAPSS) were present. The hernia was reduced and the diaphragm repaired. Neurological signs gradually resolved following surgery and 1 year postoperatively the cat was clinically normal, was not on any medication and had no evidence of hepatic dysfunction. RELEVANCE AND NOVEL INFORMATION: This is the first report of a chronic diaphragmatic rupture leading to MAPSS in a cat. SAGE Publications 2015-06-01 /pmc/articles/PMC5362922/ /pubmed/28491353 http://dx.doi.org/10.1177/2055116915585020 Text en © The Author(s) 2015 http://creativecommons.org/licenses/by-nc/3.0/ This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 3.0 License (http://www.creativecommons.org/licenses/by-nc/3.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access page(http://www.uk.sagepub.com/aboutus/openaccess.htm).
spellingShingle Case Report
Barfield, Dominic M
Gibson, Andrew D
Lipscomb, Vicky J
Multiple acquired portosystemic shunts in a cat secondary to chronic diaphragmatic rupture
title Multiple acquired portosystemic shunts in a cat secondary to chronic diaphragmatic rupture
title_full Multiple acquired portosystemic shunts in a cat secondary to chronic diaphragmatic rupture
title_fullStr Multiple acquired portosystemic shunts in a cat secondary to chronic diaphragmatic rupture
title_full_unstemmed Multiple acquired portosystemic shunts in a cat secondary to chronic diaphragmatic rupture
title_short Multiple acquired portosystemic shunts in a cat secondary to chronic diaphragmatic rupture
title_sort multiple acquired portosystemic shunts in a cat secondary to chronic diaphragmatic rupture
topic Case Report
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5362922/
https://www.ncbi.nlm.nih.gov/pubmed/28491353
http://dx.doi.org/10.1177/2055116915585020
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