Cargando…
Slit2 suppresses endothelial cell proliferation and migration by inhibiting the VEGF-Notch signaling pathway
Slit homolog 2 (Slit2) is distributed in various tissues and participates in numerous cellular processes; however, the role of Slit2 in the regulation of angiogenesis remains controversial, since it has previously been reported to exert proangiogenic and antiangiogenic activities. The present study...
Autores principales: | Li, Guo-Jian, Yang, Yong, Yang, Guo-Kai, Wan, Jia, Cui, Dao-Lei, Ma, Zhen-Huan, Du, Ling-Juan, Zhang, Gui-Min |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
D.A. Spandidos
2017
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5364956/ https://www.ncbi.nlm.nih.gov/pubmed/28260032 http://dx.doi.org/10.3892/mmr.2017.6240 |
Ejemplares similares
-
Antagonistic effects of exogenous Slit2 on VEGF-induced choroidal endothelial cell migration and tube formation
por: Tang, Yanling, et al.
Publicado: (2019) -
Effect of silencing lncRNATUG1 on rapamycin-induced inhibition of endothelial cell proliferation and migration
por: Gao, Xue, et al.
Publicado: (2018) -
Endothelial Notch signaling directly regulates the small GTPase RND1 to facilitate Notch suppression of endothelial migration
por: Swaminathan, Bhairavi, et al.
Publicado: (2022) -
Norisoboldine Suppresses VEGF-Induced Endothelial Cell Migration via the cAMP-PKA-NF-κB/Notch1 Pathway
por: Lu, Qian, et al.
Publicado: (2013) -
Notch, IL-1 and Leptin Crosstalk Outcome (NILCO) Is Critical for Leptin-Induced Proliferation, Migration and VEGF/VEGFR-2 Expression in Breast Cancer
por: Guo, Shanchun, et al.
Publicado: (2011)