Intermedin(1–53) enhances angiogenesis and attenuates adverse remodeling following myocardial infarction by activating AMP-activated protein kinase

Adverse ventricular remodeling is a maladaptive response to acute loss of myocardium and an important risk factor for heart failure following myocardial infarction (MI). Intermedin (IMD) is a novel member of the calcitonin/calcitonin gene-related peptide family, which may possess potent cardioprotec...

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Detalles Bibliográficos
Autores principales: Chen, Kankai, Yan, Meiling, Li, Yongguang, Dong, Zhifeng, Huang, Dong, Li, Jingbo, Wei, Meng
Formato: Online Artículo Texto
Lenguaje:English
Publicado: D.A. Spandidos 2017
Materias:
Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5365003/
https://www.ncbi.nlm.nih.gov/pubmed/28259938
http://dx.doi.org/10.3892/mmr.2017.6193

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5365003/
https://www.ncbi.nlm.nih.gov/pubmed/28259938
http://dx.doi.org/10.3892/mmr.2017.6193

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