Cargando…
Nicotinamide mononucleotide attenuates brain injury after intracerebral hemorrhage by activating Nrf2/HO-1 signaling pathway
Replenishment of NAD(+) has been shown to protect against brain disorders such as amyotrophic lateral sclerosis and ischemic stroke. However, whether this intervention has therapeutic effects in intracerebral hemorrhage (ICH) is unknown. In this study, we sought to determine the potential therapeuti...
Autores principales: | Wei, Chun-Chun, Kong, Yuan-Yuan, Li, Guo-Qiang, Guan, Yun-Feng, Wang, Pei, Miao, Chao-Yu |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Nature Publishing Group UK
2017
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5429727/ https://www.ncbi.nlm.nih.gov/pubmed/28386082 http://dx.doi.org/10.1038/s41598-017-00851-z |
Ejemplares similares
-
Mechanisms of transformation of nicotinamide mononucleotides to cerebral infarction hemorrhage based on MCAO model
por: Shu, Liang, et al.
Publicado: (2020) -
Antioxidant effect of nicotinamide mononucleotide in tendinopathy
por: Yamaura, Kohei, et al.
Publicado: (2022) -
Nicotinamide Mononucleotide Ameliorates Silica-Induced Lung Injury through the Nrf2-Regulated Glutathione Metabolism Pathway in Mice
por: Wang, Liqun, et al.
Publicado: (2022) -
Identification of the nicotinamide mononucleotide adenylyltransferase of
Trypanosoma cruzi
por: Niño, Carlos H, et al.
Publicado: (2015) -
Correction: Antioxidant effect of nicotinamide mononucleotide in tendinopathy
por: Yamaura, Kohei, et al.
Publicado: (2022)