Dihydroartemisinin inhibits the viability of cervical cancer cells by upregulating caveolin 1 and mitochondrial carrier homolog 2: Involvement of p53 activation and NAD(P)H:quinone oxidoreductase 1 downregulation

Dihydroartemisinin (DHA) has been shown to inhibit the viability of various cancer cells. Previous studies have revealed that the mechanisms involved in the inhibitory effects of DHA are based on theactivation of p53 and the mitochondrial-related cell death pathway. However, the exact association be...

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Detalles Bibliográficos
Autores principales: Zhang, Ting, Hu, Yuan, Wang, Ting, Cai, Peiling
Formato: Online Artículo Texto
Lenguaje:English
Publicado: D.A. Spandidos 2017
Materias:
Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5466377/
https://www.ncbi.nlm.nih.gov/pubmed/28498397
http://dx.doi.org/10.3892/ijmm.2017.2980

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5466377/
https://www.ncbi.nlm.nih.gov/pubmed/28498397
http://dx.doi.org/10.3892/ijmm.2017.2980

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