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Heterozygous eNOS Deficient Mice as a Model to Examine the Effects of eNOS Haploinsufficiency on the Cerebral Circulation
Nitric oxide derived from endothelial nitric oxide synthase (eNOS) has been shown to be a major mediator of endothelium-dependent responses in cerebral blood vessels. Loss of a single eNOS gene is not associated with any apparent negative consequences on endothelial function in most blood vessels. I...
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| Formato: | Online Artículo Texto |
| Lenguaje: | English |
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2017
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| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5467886/ https://www.ncbi.nlm.nih.gov/pubmed/28616625 |