Cargando…
TRIM32 affects the recovery of motor function following spinal cord injury through regulating proliferation of glia
Both the extrinsic environmental factors and intrinsic neuronal mechanisms limit the axonal regeneration after spinal cord injury (SCI). However, the underlying molecular mechanisms remain unclear. In the present study, we identify tripartite motif protein 32 (TRIM32), an E3 ubiquitin ligase, which...
Autores principales: | Fu, Qiang, Zou, Ming-Ming, Zhu, Jian-Wei, Zhang, Yan, Chen, Wen-Jin, Cheng, Mei, Liu, Chun-Feng, Ma, Quan-Hong, Xu, Ru-Xiang |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Impact Journals LLC
2017
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5542194/ https://www.ncbi.nlm.nih.gov/pubmed/28514764 http://dx.doi.org/10.18632/oncotarget.17492 |
Ejemplares similares
-
Deficiency of TRIM32 Impairs Motor Function and Purkinje Cells in Mid-Aged Mice
por: Zhu, Jian-Wei, et al.
Publicado: (2021) -
ASCL1 regulates proliferation of NG2‐glia in the embryonic and adult spinal cord
por: Kelenis, Demetra P., et al.
Publicado: (2018) -
TRIM32 Deficiency Impairs the Generation of Pyramidal Neurons in Developing Cerebral Cortex
por: Sun, Yan-Yun, et al.
Publicado: (2022) -
Trimming of sheep spinal cord by waterjet; an experimental study
por: Derakhshan, R., et al.
Publicado: (2023) -
NG2 Glia Reprogramming Induces Robust Axonal Regeneration After Spinal Cord Injury
por: Tai, Wenjiao, et al.
Publicado: (2023)