Cargando…
Vγ4(+)γδT Cells Aggravate Severe H1N1 Influenza Virus Infection-Induced Acute Pulmonary Immunopathological Injury via Secreting Interleukin-17A
The influenza A (H1N1) pdm09 virus remains a critical global health concern and causes high levels of morbidity and mortality. Severe acute lung injury (ALI) and acute respiratory distress syndrome (ARDS) are the major outcomes among severely infected patients. Our previous study found that interleu...
Autores principales: | Xue, Chunxue, Wen, Mingjie, Bao, Linlin, Li, Hui, Li, Fengdi, Liu, Meng, Lv, Qi, An, Yunqing, Zhang, Xulong, Cao, Bin |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Frontiers Media S.A.
2017
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5583159/ https://www.ncbi.nlm.nih.gov/pubmed/28912779 http://dx.doi.org/10.3389/fimmu.2017.01054 |
Ejemplares similares
-
Ficolin A exacerbates severe H1N1 influenza virus infection-induced acute lung immunopathological injury via excessive complement activation
por: Wu, Xu, et al.
Publicado: (2021) -
H1N1 influenza virus dose dependent induction of dysregulated innate immune responses and STAT1/3 activation are associated with pulmonary immunopathological damage
por: Yao, Duoduo, et al.
Publicado: (2022) -
Interleukin‐23 Secreted by Activated Macrophages Drives γδT Cell Production of Interleukin‐17 to Aggravate Secondary Injury After Intracerebral Hemorrhage
por: Zhong, Qi, et al.
Publicado: (2016) -
Interleukin‐17‐producing γδ T (γδ17) cells in inflammatory diseases
por: Akitsu, Aoi, et al.
Publicado: (2018) -
Delayed oseltamivir plus sirolimus treatment attenuates H1N1 virus-induced severe lung injury correlated with repressed NLRP3 inflammasome activation and inflammatory cell infiltration
por: Jia, Xuehong, et al.
Publicado: (2018)