Cargando…
PM(2.5) Exposure Elicits Oxidative Stress Responses and Mitochondrial Apoptosis Pathway Activation in HaCaT Keratinocytes
BACKGROUND: PM(2.5) (aerodynamic diameter ≤ 2.5 μm) is a dominant and ubiquitous air pollutant that has become a global concern as PM(2.5) exposure has been linked to many adverse health effects including cardiovascular and pulmonary diseases. Emerging evidence supports a correlation between increas...
Autores principales: | Hu, Rong, Xie, Xiao-Yuan, Xu, Si-Ka, Wang, Ya-Ning, Jiang, Ming, Wen, Li-Rong, Lai, Wei, Guan, Lei |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Medknow Publications & Media Pvt Ltd
2017
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5598333/ https://www.ncbi.nlm.nih.gov/pubmed/28816208 http://dx.doi.org/10.4103/0366-6999.212942 |
Ejemplares similares
-
The cytotoxic mechanism of epigallocatechin gallate on proliferative HaCaT keratinocytes
por: Chu, Yu-Wen, et al.
Publicado: (2017) -
Melanoma stimulates the proteolytic activity of HaCaT keratinocytes
por: Mazurkiewicz, Justyna, et al.
Publicado: (2022) -
Effects of Ambient Fine Particles PM(2.5) on Human HaCaT Cells
por: Li, Qiao, et al.
Publicado: (2017) -
Protein dataset of immortalized keratinocyte HaCaT cells and normal human keratinocytes
por: Rusanov, A.L., et al.
Publicado: (2021) -
Cytotoxicity patterns of arsenic trioxide exposure on HaCaT keratinocytes
por: Udensi, Udensi K, et al.
Publicado: (2011)