Cargando…
A cell cycle-independent mode of the Rad9-Dpb11 interaction is induced by DNA damage
Budding yeast Rad9, like its orthologs, controls two aspects of the cellular response to DNA double strand breaks (DSBs) – signalling of the DNA damage checkpoint and DNA end resection. Rad9 binds to damaged chromatin via modified nucleosomes independently of the cell cycle phase. Additionally, Rad9...
Autores principales: | di Cicco, Giulia, Bantele, Susanne C. S., Reusswig, Karl-Uwe, Pfander, Boris |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Nature Publishing Group UK
2017
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5599684/ https://www.ncbi.nlm.nih.gov/pubmed/28912563 http://dx.doi.org/10.1038/s41598-017-11937-z |
Ejemplares similares
-
Dpb11 coordinates Mec1 kinase activation with cell cycle-regulated Rad9 recruitment
por: Pfander, Boris, et al.
Publicado: (2011) -
Targeting of the Fun30 nucleosome remodeller by the Dpb11 scaffold facilitates cell cycle-regulated DNA end resection
por: Bantele, Susanne CS, et al.
Publicado: (2017) -
A cell cycle-regulated Slx4–Dpb11 complex promotes the resolution of DNA repair intermediates linked to stalled replication
por: Gritenaite, Dalia, et al.
Publicado: (2014) -
Quantitative mechanisms of DNA damage sensing and signaling
por: Bantele, Susanne C. S., et al.
Publicado: (2019) -
Nucleosome Remodeling by Fun30(SMARCAD1) in the DNA Damage Response
por: Bantele, Susanne C. S., et al.
Publicado: (2019)