Cargando…
MAZ induces MYB expression during the exit from quiescence via the E2F site in the MYB promoter
Most E2F-binding sites repress transcription through the recruitment of Retinoblastoma (RB) family members until the end of the G1 cell-cycle phase. Although the MYB promoter contains an E2F-binding site, its transcription is activated shortly after the exit from quiescence, before RB family members...
Autores principales: | Álvaro-Blanco, Josué, Urso, Katia, Chiodo, Yuri, Martín-Cortázar, Carla, Kourani, Omar, Arco, Pablo Gómez-del, Rodríguez-Martínez, María, Calonge, Esther, Alcamí, José, Redondo, Juan Miguel, Iglesias, Teresa, Campanero, Miguel R. |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Oxford University Press
2017
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5622404/ https://www.ncbi.nlm.nih.gov/pubmed/28973440 http://dx.doi.org/10.1093/nar/gkx641 |
Ejemplares similares
-
Specificity of MYB interactions relies on motifs in ordered and disordered contexts
por: Millard, Peter S, et al.
Publicado: (2019) -
The Myb/SANT domain of the telomere-binding protein TRF2 alters chromatin structure
por: Baker, Asmaa M., et al.
Publicado: (2009) -
A Myc–microRNA network promotes exit from quiescence by suppressing the interferon response and cell-cycle arrest genes
por: Polioudakis, Damon, et al.
Publicado: (2013) -
Estrogen receptor-α recruits P-TEFb to overcome transcriptional pausing in intron 1 of the MYB gene
por: Mitra, Partha, et al.
Publicado: (2012) -
Activation of the oncogenic transcription factor B-Myb via multisite phosphorylation and prolyl cis/trans isomerization
por: Werwein, Eugen, et al.
Publicado: (2019)