Cargando…
Glucose Limitation Alters Glutamine Metabolism in MUC1-Overexpressing Pancreatic Cancer Cells
[Image: see text] Pancreatic cancer cells overexpressing Mucin 1 (MUC1) rely on aerobic glycolysis and, correspondingly, are dependent on glucose for survival. Our NMR metabolomics comparative analysis of control (S2–013.Neo) and MUC1-overexpressing (S2–013.MUC1) cells demonstrates that MUC1 reprogr...
Autores principales: | Gebregiworgis, Teklab, Purohit, Vinee, Shukla, Surendra K., Tadros, Saber, Chaika, Nina V., Abrego, Jaime, Mulder, Scott E., Gunda, Venugopal, Singh, Pankaj K., Powers, Robert |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
American Chemical
Society
2017
|
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5634392/ https://www.ncbi.nlm.nih.gov/pubmed/28809118 http://dx.doi.org/10.1021/acs.jproteome.7b00246 |
Ejemplares similares
-
Correction: Metabolic reprogramming induced by ketone bodies diminishes pancreatic cancer cachexia
por: Shukla, Surendra K, et al.
Publicado: (2014) -
Metabolic reprogramming induced by ketone bodies diminishes pancreatic cancer cachexia
por: Shukla, Surendra K, et al.
Publicado: (2014) -
MUC1 facilitates metabolomic reprogramming in triple-negative breast cancer
por: Goode, Gennifer, et al.
Publicado: (2017) -
Correction: MUC1 facilitates metabolomic reprogramming in triple-negative breast cancer
por: Goode, Gennifer, et al.
Publicado: (2017) -
MUC16-mediated activation of mTOR and c-MYC reprograms pancreatic cancer metabolism
por: Shukla, Surendra K., et al.
Publicado: (2015)