Cargando…

Differentiation of IL-17-Producing Invariant Natural Killer T Cells Requires Expression of the Transcription Factor c-Maf

c-Maf belongs to the large Maf family of transcription factors and plays a key role in the regulation of cytokine production and differentiation of T(H)2, T(H)17, T(FH), and Tr1 cells. Invariant natural killer T (iNKT) cells can rapidly produce large quantity of T(H)-related cytokines such as IFN-γ,...

Descripción completa

Detalles Bibliográficos
Autores principales:	Yu, Jhang-Sian, Hamada, Michito, Ohtsuka, Shigeo, Yoh, Keigyou, Takahashi, Satoru, Miaw, Shi-Chuen
Formato:	Online Artículo Texto
Lenguaje:	English
Publicado:	Frontiers Media S.A. 2017
Materias:	Immunology
Acceso en línea:	https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5663684/ https://www.ncbi.nlm.nih.gov/pubmed/29163480 http://dx.doi.org/10.3389/fimmu.2017.01399

Ejemplares similares

Role of MafB in macrophages
por: Hamada, Michito, et al.
Publicado: (2019)

Th2-biased GATA-3 transgenic mice developed severe experimental peritoneal fibrosis compared with Th1-biased T-bet and Th17-biased RORγt transgenic mice
por: Yoh, Keigyou, et al.
Publicado: (2015)

MafB deficiency accelerates the development of obesity in mice
por: Tran, Mai Thi Nhu, et al.
Publicado: (2016)

The transcription factor c-Maf is essential for the commitment of IL-17-producing γδ T cells
por: Zuberbuehler, Matthew K., et al.
Publicado: (2018)

MafB Maintains β-Cell Identity under MafA-Deficient Conditions
por: Deng, Zhaobin, et al.
Publicado: (2022)

Development and Function of Invariant Natural Killer T Cells Producing T(H)2- and T(H)17-Cytokines
por: Watarai, Hiroshi, et al.
Publicado: (2012)

Longitudinal analysis of invariant natural killer T cell activation reveals a cMAF-associated transcriptional state of NKT10 cells
por: Kane, Harry, et al.
Publicado: (2022)

Overexpression of GATA-3 in T Cells Accelerates Dextran Sulfate Sodium-Induced Colitis
por: Okamura, Midori, et al.
Publicado: (2014)

MafB is a critical regulator of complement component C1q
por: Tran, Mai Thi Nhu, et al.
Publicado: (2017)

MafA Is Required for Postnatal Proliferation of Pancreatic β-Cells
por: Eto, Koki, et al.
Publicado: (2014)

MafB Is Important for Pancreatic β-Cell Maintenance under a MafA-Deficient Condition
por: Xiafukaiti, Gulibaikelamu, et al.
Publicado: (2019)

An Inducible Diabetes Mellitus Murine Model Based on MafB Conditional Knockout under MafA-Deficient Condition
por: Deng, Zhaobin, et al.
Publicado: (2020)

Reciprocal Regulation of C-Maf Tyrosine Phosphorylation by Tec and Ptpn22
por: Liu, Chih-Chun, et al.
Publicado: (2015)

Radiation inducible MafB gene is required for thymic regeneration
por: Hashimoto, Daiki, et al.
Publicado: (2021)

Functional analysis of large MAF transcription factors and elucidation of their relationships with human diseases
por: Takahashi, Satoru
Publicado: (2021)

Increased IL-22- and IL-17A-Producing Mucosal-Associated Invariant T Cells in the Peripheral Blood of Patients With Ankylosing Spondylitis
por: Toussirot, Éric, et al.
Publicado: (2018)

T-bet and Eomes instruct the development of two distinct natural killer cell lineages in the liver and in the bone marrow
por: Daussy, Cécile, et al.
Publicado: (2014)

Exploring Large MAF Transcription Factors: Functions, Pathology, and Mouse Models with Point Mutations
por: Fujino, Mitsunori, et al.
Publicado: (2023)

Invariant Natural Killer T and Mucosal-Associated Invariant T Cells in Asthmatic Patients
por: Lezmi, Guillaume, et al.
Publicado: (2018)

IL-17 and IL-17-producing cells in protection versus pathology
por: Mills, Kingston H. G.
Publicado: (2022)

Loss of MafA and MafB expression promotes islet inflammation
por: Singh, Tania, et al.
Publicado: (2019)

Role of large MAF transcription factors in the mouse endocrine pancreas
por: ABDELLATIF, Ahmed M., et al.
Publicado: (2015)

Overexpression of RORγt Enhances Pulmonary Inflammation after Infection with Mycobacterium Avium
por: Matsuyama, Masashi, et al.
Publicado: (2016)

T-bet, but not Gata3, overexpression is detrimental in a neurotropic viral infection
por: Sato, Fumitaka, et al.
Publicado: (2017)

IL-27 triggers IL-10 production in Th17 cells via a c-Maf/RORγt/Blimp-1 signal to promote the progression of endometriosis
por: Chang, Kai-Kai, et al.
Publicado: (2017)

Combinatorial motif analysis of regulatory gene expression in Mafb deficient macrophages
por: Morita, Mariko, et al.
Publicado: (2011)

Molecular Mechanisms Driving IL-10- Producing B Cells Functions: STAT3 and c-MAF as Underestimated Central Key Regulators?
por: Michée-Cospolite, Magalie, et al.
Publicado: (2022)

Antitumor Responses of Invariant Natural Killer T Cells
por: Altman, Jennie B., et al.
Publicado: (2015)

Immunometabolic Activation of Invariant Natural Killer T Cells
por: Ververs, Francesca A., et al.
Publicado: (2018)

Antigen specificity of invariant natural killer T-cells
por: Birkholz, Alysia M., et al.
Publicado: (2015)

The two faces of invariant natural killer T cells
por: Walton, Emma L.
Publicado: (2015)

Metabolism in Invariant Natural Killer T Cells: An Overview
por: Yarosz, Emily L., et al.
Publicado: (2021)

Invariant natural killer T cells in lung diseases
por: Jeong, Dongjin, et al.
Publicado: (2023)

MafK accelerates Salmonella mucosal infection through caspase-3 activation
por: Xu, Shiyao, et al.
Publicado: (2022)

HEB is required for the specification of fetal IL-17-producing γδ T cells
por: In, Tracy S. H., et al.
Publicado: (2017)

Maf deficiency in T cells dysregulates T(reg) - T(H)17 balance leading to spontaneous colitis
por: Imbratta, Claire, et al.
Publicado: (2019)

Th17 cells are not required for maintenance of IL-17A producing γδ T cells in vivo
por: Gupta, Pawan K., et al.
Publicado: (2016)

Antigen-driven bystander effect accelerates epicutaneous sensitization with a new protein allergen
por: Wang, Li-Fang, et al.
Publicado: (2009)

Regulation of an Autoimmune Model for Multiple Sclerosis in Th2-Biased GATA3 Transgenic Mice
por: Fernando, Viromi, et al.
Publicado: (2014)

IL-17 Production from T Helper 17, Mucosal-Associated Invariant T, and γδ Cells in Tuberculosis Infection and Disease
por: Coulter, Felicity, et al.
Publicado: (2017)

Cannot write session to /tmp/vufind_sessions/sess_93kdtu2o2a7ie0sug20ldqh9je