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A novel DNA damage-induced alternative splicing pathway that regulates p53 and cellular senescence markers

Detalles Bibliográficos
Autores principales: Chen, Jing, Kastan, Michael B.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Impact Journals LLC 2017
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5672893/
https://www.ncbi.nlm.nih.gov/pubmed/29142900
http://dx.doi.org/10.18632/oncoscience.367
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author Chen, Jing
Kastan, Michael B.
author_facet Chen, Jing
Kastan, Michael B.
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spelling pubmed-56728932017-11-15 A novel DNA damage-induced alternative splicing pathway that regulates p53 and cellular senescence markers Chen, Jing Kastan, Michael B. Oncoscience Editorial Impact Journals LLC 2017-10-12 /pmc/articles/PMC5672893/ /pubmed/29142900 http://dx.doi.org/10.18632/oncoscience.367 Text en Copyright: © 2017 Chen and Kastan http://creativecommons.org/licenses/by/3.0/ This article is distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/3.0/) (CC-BY), which permits unrestricted use and redistribution provided that the original author and source are credited.
spellingShingle Editorial
Chen, Jing
Kastan, Michael B.
A novel DNA damage-induced alternative splicing pathway that regulates p53 and cellular senescence markers
title A novel DNA damage-induced alternative splicing pathway that regulates p53 and cellular senescence markers
title_full A novel DNA damage-induced alternative splicing pathway that regulates p53 and cellular senescence markers
title_fullStr A novel DNA damage-induced alternative splicing pathway that regulates p53 and cellular senescence markers
title_full_unstemmed A novel DNA damage-induced alternative splicing pathway that regulates p53 and cellular senescence markers
title_short A novel DNA damage-induced alternative splicing pathway that regulates p53 and cellular senescence markers
title_sort novel dna damage-induced alternative splicing pathway that regulates p53 and cellular senescence markers
topic Editorial
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5672893/
https://www.ncbi.nlm.nih.gov/pubmed/29142900
http://dx.doi.org/10.18632/oncoscience.367
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