Cargando…
Traumatic brain injury is unlikely precipitating Leigh syndrome due to the GJB2 mutation c.35delG
Autores principales: | Finsterer, Josef, Zarrouk-Mahjoub, Sinda |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Shahid Beheshti University of Medical Sciences
2017
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5703751/ https://www.ncbi.nlm.nih.gov/pubmed/29201956 |
Ejemplares similares
-
Vaccination triggering onset of m.8993T > G associated Leigh syndrome
por: Finsterer, Josef, et al.
Publicado: (2018) -
Why does Leigh syndrome respond to immunotherapy?
por: Finsterer, Josef, et al.
Publicado: (2016) -
Transient symmetric T2-hyperintensities of basal ganglia and brainstem not only point to Leigh syndrome
por: Finsterer, Josef, et al.
Publicado: (2016) -
Updated carrier rates for c.35delG (GJB2) associated with hearing loss in Russia and common c.35delG haplotypes in Siberia
por: Zytsar, Marina V., et al.
Publicado: (2018) -
Mitochondrial vasculopathy due to the m.3243A > G mutation is not restricted to the carotid artery
por: Finsterer, Josef, et al.
Publicado: (2016)