Cargando…
Lipocalin-2 in Fructose-Induced Fatty Liver Disease
The intake of excess dietary fructose most often leads to non-alcoholic fatty liver disease (NAFLD). Fructose is metabolized mainly in the liver and its chronic consumption results in lipogenic gene expression in this organ. However, precisely how fructose is involved in NAFLD progression is still n...
Autores principales: | Lambertz, Jessica, Berger, Thorsten, Mak, Tak W., van Helden, Josef, Weiskirchen, Ralf |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Frontiers Media S.A.
2017
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5712346/ https://www.ncbi.nlm.nih.gov/pubmed/29234288 http://dx.doi.org/10.3389/fphys.2017.00964 |
Ejemplares similares
-
Fructose: A Dietary Sugar in Crosstalk with Microbiota Contributing to the Development and Progression of Non-Alcoholic Liver Disease
por: Lambertz, Jessica, et al.
Publicado: (2017) -
Lipocalin 2 (LCN2) Expression in Hepatic Malfunction and Therapy
por: Asimakopoulou, Anastasia, et al.
Publicado: (2016) -
Quantification of Short Chain Fatty Acids (acetate, butyrate, propionate) in human blood with ion exclusion chromatography
por: Bertges, Markus, et al.
Publicado: (2021) -
Tear Lipocalin and Lipocalin-Interacting Membrane Receptor
por: Glasgow, Ben J.
Publicado: (2021) -
Editorial: Liver Myofibroblasts
por: Kanta, Jiri, et al.
Publicado: (2016)