Leydig cell tumor of the testis, presenting with azoospermia

CASE: A case of Leydig cell tumor, associated with azoospermia, is presented. OUTCOME: The levels of sex hormones obviously were decreased, including luteinizing hormone (LH) and follicle‐stimulating hormone (FSH), with elevated testosterone. Computed tomography revealed no adrenal gland tumor, but...

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Detalles Bibliográficos
Autores principales: Hibi, Hatsuki, Yamashita, Kyoko, Sumitomo, Makoto, Asada, Yoshimasa
Formato: Online Artículo Texto
Lenguaje:English
Publicado: John Wiley and Sons Inc. 2017
Materias:
Case Reports
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5715892/
https://www.ncbi.nlm.nih.gov/pubmed/29259494
http://dx.doi.org/10.1002/rmb2.12046
Descripción
Sumario:CASE: A case of Leydig cell tumor, associated with azoospermia, is presented. OUTCOME: The levels of sex hormones obviously were decreased, including luteinizing hormone (LH) and follicle‐stimulating hormone (FSH), with elevated testosterone. Computed tomography revealed no adrenal gland tumor, but a significant calcification in the right scrotal content was observed. He received a right radical orchiectomy and then he was unable to ejaculate. An endocrine panel revealed significantly decreased levels of testosterone and the low LH level had remained. Hormone replacement therapy with combined LH and FSH successfully recovered and preserved spermatogenesis. CONCLUSIONS: Although the patient’s sexual function deteriorated after surgery, hormone replacement therapy was successful in establishing spermatogenesis.

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