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Data supporting the angiotensin II activates MEL18 to deSUMOylate HSF2 for hypertension-related heart failure
In association with the published article “Inhibition of HSF2 SUMOylation via MEL18 upregulates IGF-IIR and leads to hypertension-induced cardiac hypertrophy” (Huang et al., 2017) [1], this data article contains information about deSUMOylation of HSF2 on lysine 82 on angiotensin II (ANG II) -induced...
Autores principales: | , , , , , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Elsevier
2017
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5734702/ https://www.ncbi.nlm.nih.gov/pubmed/29270451 http://dx.doi.org/10.1016/j.dib.2017.11.040 |
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author | Huang, Chih-Yang Kuo, Chia-Hua Pai, Pei-Ying Ho, Tsung-Jung Lin, Yueh-Min Chen, Ray-Jade Tsai, Fuu-Jen Vijaya Padma, V. Kuo, Wei-Wen Huang, Chih-Yang |
author_facet | Huang, Chih-Yang Kuo, Chia-Hua Pai, Pei-Ying Ho, Tsung-Jung Lin, Yueh-Min Chen, Ray-Jade Tsai, Fuu-Jen Vijaya Padma, V. Kuo, Wei-Wen Huang, Chih-Yang |
author_sort | Huang, Chih-Yang |
collection | PubMed |
description | In association with the published article “Inhibition of HSF2 SUMOylation via MEL18 upregulates IGF-IIR and leads to hypertension-induced cardiac hypertrophy” (Huang et al., 2017) [1], this data article contains information about deSUMOylation of HSF2 on lysine 82 on angiotensin II (ANG II) -induced cardiac hypertrophy, which is mediated by MEL18. Isolated adult human whole heart tissue showed MEL18-mediated HSF2-IGF-IIR pathway is upregulated in hypertension human heart, compared to health human heart. |
format | Online Article Text |
id | pubmed-5734702 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2017 |
publisher | Elsevier |
record_format | MEDLINE/PubMed |
spelling | pubmed-57347022017-12-21 Data supporting the angiotensin II activates MEL18 to deSUMOylate HSF2 for hypertension-related heart failure Huang, Chih-Yang Kuo, Chia-Hua Pai, Pei-Ying Ho, Tsung-Jung Lin, Yueh-Min Chen, Ray-Jade Tsai, Fuu-Jen Vijaya Padma, V. Kuo, Wei-Wen Huang, Chih-Yang Data Brief Medicine and Dentistry In association with the published article “Inhibition of HSF2 SUMOylation via MEL18 upregulates IGF-IIR and leads to hypertension-induced cardiac hypertrophy” (Huang et al., 2017) [1], this data article contains information about deSUMOylation of HSF2 on lysine 82 on angiotensin II (ANG II) -induced cardiac hypertrophy, which is mediated by MEL18. Isolated adult human whole heart tissue showed MEL18-mediated HSF2-IGF-IIR pathway is upregulated in hypertension human heart, compared to health human heart. Elsevier 2017-11-20 /pmc/articles/PMC5734702/ /pubmed/29270451 http://dx.doi.org/10.1016/j.dib.2017.11.040 Text en © 2018 Published by Elsevier Inc. http://creativecommons.org/licenses/by/4.0/ This is an open access article under the CC BY license (http://creativecommons.org/licenses/by/4.0/). |
spellingShingle | Medicine and Dentistry Huang, Chih-Yang Kuo, Chia-Hua Pai, Pei-Ying Ho, Tsung-Jung Lin, Yueh-Min Chen, Ray-Jade Tsai, Fuu-Jen Vijaya Padma, V. Kuo, Wei-Wen Huang, Chih-Yang Data supporting the angiotensin II activates MEL18 to deSUMOylate HSF2 for hypertension-related heart failure |
title | Data supporting the angiotensin II activates MEL18 to deSUMOylate HSF2 for hypertension-related heart failure |
title_full | Data supporting the angiotensin II activates MEL18 to deSUMOylate HSF2 for hypertension-related heart failure |
title_fullStr | Data supporting the angiotensin II activates MEL18 to deSUMOylate HSF2 for hypertension-related heart failure |
title_full_unstemmed | Data supporting the angiotensin II activates MEL18 to deSUMOylate HSF2 for hypertension-related heart failure |
title_short | Data supporting the angiotensin II activates MEL18 to deSUMOylate HSF2 for hypertension-related heart failure |
title_sort | data supporting the angiotensin ii activates mel18 to desumoylate hsf2 for hypertension-related heart failure |
topic | Medicine and Dentistry |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5734702/ https://www.ncbi.nlm.nih.gov/pubmed/29270451 http://dx.doi.org/10.1016/j.dib.2017.11.040 |
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