Cargando…
IL-33 promotes the egress of group 2 innate lymphoid cells from the bone marrow
Group 2 innate lymphoid cells (ILC2s) are effector cells within the mucosa and key participants in type 2 immune responses in the context of allergic inflammation and infection. ILC2s develop in the bone marrow from common lymphoid progenitor cells, but little is known about how ILC2s egress from th...
Autores principales: | Stier, Matthew T., Zhang, Jian, Goleniewska, Kasia, Cephus, Jacqueline Y., Rusznak, Mark, Wu, Lan, Van Kaer, Luc, Zhou, Baohua, Newcomb, Dawn C., Peebles, R. Stokes |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
The Rockefeller University Press
2018
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5748848/ https://www.ncbi.nlm.nih.gov/pubmed/29222107 http://dx.doi.org/10.1084/jem.20170449 |
Ejemplares similares
-
Prostaglandin I2 inhibits IL-33-induced IL-5 and IL-13 production by human type 2 innate lymphoid cells
por: Zhou, Weisong, et al.
Publicado: (2015) -
Respiratory syncytial virus infection activates IL-13–producing group 2 innate lymphoid cells through thymic stromal lymphopoietin
por: Stier, Matthew T., et al.
Publicado: (2016) -
TSLP and IL‐33 reciprocally promote each other's lung protein expression and ILC2 receptor expression to enhance innate type‐2 airway inflammation
por: Toki, Shinji, et al.
Publicado: (2020) -
The histone deacetylase inhibitor trichostatin A suppresses murine innate allergic inflammation by blocking group 2 innate lymphoid cell (ILC2) activation
por: Toki, Shinji, et al.
Publicado: (2016) -
Development and function of regulatory innate lymphoid cells
por: Thomas, Christopher M., et al.
Publicado: (2022)