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Zinc status is associated with inflammation, oxidative stress, lipid, and glucose metabolism

A number of studies have reported that zinc plays a substantial role in the development of metabolic syndrome, taking part in the regulation of cytokine expression, suppressing inflammation, and is also required to activate antioxidant enzymes that scavenge reactive oxygen species, reducing oxidativ...

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Detalles Bibliográficos
Autores principales: Olechnowicz, J., Tinkov, A., Skalny, A., Suliburska, Joanna
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Springer Japan 2017
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5754376/
https://www.ncbi.nlm.nih.gov/pubmed/28965330
http://dx.doi.org/10.1007/s12576-017-0571-7
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author Olechnowicz, J.
Tinkov, A.
Skalny, A.
Suliburska, Joanna
author_facet Olechnowicz, J.
Tinkov, A.
Skalny, A.
Suliburska, Joanna
author_sort Olechnowicz, J.
collection PubMed
description A number of studies have reported that zinc plays a substantial role in the development of metabolic syndrome, taking part in the regulation of cytokine expression, suppressing inflammation, and is also required to activate antioxidant enzymes that scavenge reactive oxygen species, reducing oxidative stress. Zinc also plays a role in the correct functioning of lipid and glucose metabolism, regulating and forming the expression of insulin. In numerous studies, zinc supplementation has been found to improve blood pressure, glucose, and LDL cholesterol serum level. Deeper knowledge of zinc’s properties may help in treating metabolic syndrome, thus protecting against stroke and angina pectoris, and ultimately against death.
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spelling pubmed-57543762018-01-22 Zinc status is associated with inflammation, oxidative stress, lipid, and glucose metabolism Olechnowicz, J. Tinkov, A. Skalny, A. Suliburska, Joanna J Physiol Sci Review A number of studies have reported that zinc plays a substantial role in the development of metabolic syndrome, taking part in the regulation of cytokine expression, suppressing inflammation, and is also required to activate antioxidant enzymes that scavenge reactive oxygen species, reducing oxidative stress. Zinc also plays a role in the correct functioning of lipid and glucose metabolism, regulating and forming the expression of insulin. In numerous studies, zinc supplementation has been found to improve blood pressure, glucose, and LDL cholesterol serum level. Deeper knowledge of zinc’s properties may help in treating metabolic syndrome, thus protecting against stroke and angina pectoris, and ultimately against death. Springer Japan 2017-09-30 2018 /pmc/articles/PMC5754376/ /pubmed/28965330 http://dx.doi.org/10.1007/s12576-017-0571-7 Text en © The Author(s) 2017 Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made.
spellingShingle Review
Olechnowicz, J.
Tinkov, A.
Skalny, A.
Suliburska, Joanna
Zinc status is associated with inflammation, oxidative stress, lipid, and glucose metabolism
title Zinc status is associated with inflammation, oxidative stress, lipid, and glucose metabolism
title_full Zinc status is associated with inflammation, oxidative stress, lipid, and glucose metabolism
title_fullStr Zinc status is associated with inflammation, oxidative stress, lipid, and glucose metabolism
title_full_unstemmed Zinc status is associated with inflammation, oxidative stress, lipid, and glucose metabolism
title_short Zinc status is associated with inflammation, oxidative stress, lipid, and glucose metabolism
title_sort zinc status is associated with inflammation, oxidative stress, lipid, and glucose metabolism
topic Review
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5754376/
https://www.ncbi.nlm.nih.gov/pubmed/28965330
http://dx.doi.org/10.1007/s12576-017-0571-7
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