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Deletion of the alpha-synuclein locus in a subpopulation of C57BL/6J inbred mice
BACKGROUND: The presynaptic protein α-synuclein is involved in a range of neurodegenerative diseases. Here we analyze potential compensatory mechanisms in α-synuclein null mutant mice. Furthermore, the findings reveal problems that may be associated with inbred mouse strains. RESULTS: Expression pro...
| Autores principales: | , |
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| Formato: | Texto |
| Lenguaje: | English |
| Publicado: |
BioMed Central
2001
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| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC57740/ https://www.ncbi.nlm.nih.gov/pubmed/11591219 http://dx.doi.org/10.1186/1471-2202-2-11 |