Suppression of Arrhythmia by Enhancing Mitochondrial Ca(2+) Uptake in Catecholaminergic Ventricular Tachycardia Models

Cardiovascular disease-related deaths frequently arise from arrhythmias, but treatment options are limited due to perilous side effects of commonly used antiarrhythmic drugs. Cardiac rhythmicity strongly depends on cardiomyocyte Ca(2+) handling and prevalent cardiac diseases are causally associated...

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Detalles Bibliográficos
Autores principales: Schweitzer, Maria K., Wilting, Fabiola, Sedej, Simon, Dreizehnter, Lisa, Dupper, Nathan J., Tian, Qinghai, Moretti, Alessandra, My, Ilaria, Kwon, Ohyun, Priori, Silvia G., Laugwitz, Karl-Ludwig, Storch, Ursula, Lipp, Peter, Breit, Andreas, Mederos y Schnitzler, Michael, Gudermann, Thomas, Schredelseker, Johann
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Elsevier 2017
Materias:
PRECLINICAL RESEARCH
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5774336/
https://www.ncbi.nlm.nih.gov/pubmed/29354781
http://dx.doi.org/10.1016/j.jacbts.2017.06.008

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