Cargando…
Suppression of CHRN endocytosis by carbonic anhydrase CAR3 in the pathogenesis of myasthenia gravis
Myasthenia gravis is an autoimmune disorder of the neuromuscular junction manifested as fatigable muscle weakness, which is typically caused by pathogenic autoantibodies against postsynaptic CHRN/AChR (cholinergic receptor nicotinic) in the endplate of skeletal muscle. Our previous studies have iden...
Autores principales: | Du, Ailian, Huang, Shiqian, Zhao, Xiaonan, Feng, Kuan, Zhang, Shuangyan, Huang, Jiefang, Miao, Xiang, Baggi, Fulvio, Ostrom, Rennolds S., Zhang, Yanyun, Chen, Xiangjun, Xu, Congfeng |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Taylor & Francis
2017
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5788490/ https://www.ncbi.nlm.nih.gov/pubmed/28933591 http://dx.doi.org/10.1080/15548627.2017.1375633 |
Ejemplares similares
-
Animal models of myasthenia gravis: utility and limitations
por: Mantegazza, Renato, et al.
Publicado: (2016) -
Editorial: Phenotypes of myasthenia gravis
por: Li, Hai-Feng, et al.
Publicado: (2022) -
Therapeutic Effect of Bifidobacterium Administration on Experimental Autoimmune Myasthenia Gravis in Lewis Rats
por: Rinaldi, Elena, et al.
Publicado: (2019) -
Immunoblot analysis of antiAChR antibodies in myasthenia gravis
por: Mantegazza, R., et al.
Publicado: (1988) -
Administration of bifidobacterium and lactobacillus strains modulates experimental myasthenia gravis and experimental encephalomyelitis in Lewis rats
por: Consonni, Alessandra, et al.
Publicado: (2018)