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Pulmonary hypertension in stiff left atrial syndrome: pathogenesis and treatment in one

We present the rare case of a patient with pulmonary hypertension in the context of the stiff left atrial syndrome after extensive catheter ablation, a unique constellation characterized by high pulmonary artery and pulmonary artery wedge pressures due to left atrial dysfunction but normal left vent...

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Autores principales: Maeder, Micha T., Nägele, Reto, Rohner, Philipp, Weilenmann, Daniel
Formato: Online Artículo Texto
Lenguaje:English
Publicado: John Wiley and Sons Inc. 2017
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5793980/
https://www.ncbi.nlm.nih.gov/pubmed/29115016
http://dx.doi.org/10.1002/ehf2.12234
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author Maeder, Micha T.
Nägele, Reto
Rohner, Philipp
Weilenmann, Daniel
author_facet Maeder, Micha T.
Nägele, Reto
Rohner, Philipp
Weilenmann, Daniel
author_sort Maeder, Micha T.
collection PubMed
description We present the rare case of a patient with pulmonary hypertension in the context of the stiff left atrial syndrome after extensive catheter ablation, a unique constellation characterized by high pulmonary artery and pulmonary artery wedge pressures due to left atrial dysfunction but normal left ventricular end‐diastolic pressure, normal mitral valve, and absence pulmonary vein stenosis. This patient was surprisingly oligosymptomatic, however, which may have been due to a persistent post‐puncture atrial septal defect, which may have allowed for controlled left atrial decompression, which is in line with the novel concept of the catheter‐based creation of an intracardiac shunt as a treatment for heart failure.
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spelling pubmed-57939802018-02-14 Pulmonary hypertension in stiff left atrial syndrome: pathogenesis and treatment in one Maeder, Micha T. Nägele, Reto Rohner, Philipp Weilenmann, Daniel ESC Heart Fail Case Reports We present the rare case of a patient with pulmonary hypertension in the context of the stiff left atrial syndrome after extensive catheter ablation, a unique constellation characterized by high pulmonary artery and pulmonary artery wedge pressures due to left atrial dysfunction but normal left ventricular end‐diastolic pressure, normal mitral valve, and absence pulmonary vein stenosis. This patient was surprisingly oligosymptomatic, however, which may have been due to a persistent post‐puncture atrial septal defect, which may have allowed for controlled left atrial decompression, which is in line with the novel concept of the catheter‐based creation of an intracardiac shunt as a treatment for heart failure. John Wiley and Sons Inc. 2017-11-08 /pmc/articles/PMC5793980/ /pubmed/29115016 http://dx.doi.org/10.1002/ehf2.12234 Text en © 2017 The Authors. ESC Heart Failure published by John Wiley & Sons Ltd on behalf of the European Society of Cardiology. This is an open access article under the terms of the Creative Commons Attribution‐NonCommercial‐NoDerivs (http://creativecommons.org/licenses/by-nc-nd/4.0/) License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non‐commercial and no modifications or adaptations are made.
spellingShingle Case Reports
Maeder, Micha T.
Nägele, Reto
Rohner, Philipp
Weilenmann, Daniel
Pulmonary hypertension in stiff left atrial syndrome: pathogenesis and treatment in one
title Pulmonary hypertension in stiff left atrial syndrome: pathogenesis and treatment in one
title_full Pulmonary hypertension in stiff left atrial syndrome: pathogenesis and treatment in one
title_fullStr Pulmonary hypertension in stiff left atrial syndrome: pathogenesis and treatment in one
title_full_unstemmed Pulmonary hypertension in stiff left atrial syndrome: pathogenesis and treatment in one
title_short Pulmonary hypertension in stiff left atrial syndrome: pathogenesis and treatment in one
title_sort pulmonary hypertension in stiff left atrial syndrome: pathogenesis and treatment in one
topic Case Reports
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5793980/
https://www.ncbi.nlm.nih.gov/pubmed/29115016
http://dx.doi.org/10.1002/ehf2.12234
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