Cargando…
Complement Factor H Inhibits Anti-Neutrophil Cytoplasmic Autoantibody-Induced Neutrophil Activation by Interacting With Neutrophils
Our previous study demonstrated that plasma levels of complement factor H (FH) were inversely associated with the disease activity of patients with anti-neutrophil cytoplasmic autoantibody (ANCA)-associated vasculitis (AAV). In addition to serving as an inhibitor of the alternative complement pathwa...
Autores principales: | Chen, Su-Fang, Wang, Feng-Mei, Li, Zhi-Ying, Yu, Feng, Chen, Min, Zhao, Ming-Hui |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Frontiers Media S.A.
2018
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5867335/ https://www.ncbi.nlm.nih.gov/pubmed/29616045 http://dx.doi.org/10.3389/fimmu.2018.00559 |
Ejemplares similares
-
Pathogenetic and Clinical Aspects of Anti-Neutrophil Cytoplasmic Autoantibody-Associated Vasculitides
por: Lamprecht, Peter, et al.
Publicado: (2018) -
Neutrophil Extracellular Traps Contain Selected Antigens of Anti-Neutrophil Cytoplasmic Antibodies
por: Panda, Rachita, et al.
Publicado: (2017) -
Digital Spatial Profiling of Individual Glomeruli From Patients With Anti-Neutrophil Cytoplasmic Autoantibody-Associated Glomerulonephritis
por: Ye, Lin, et al.
Publicado: (2022) -
Influence of variable domain glycosylation on anti-neutrophil cytoplasmic autoantibodies and anti-glomerular basement membrane autoantibodies
por: Xu, Peng-Cheng, et al.
Publicado: (2012) -
Neutrophil activation in patients with anti-neutrophil cytoplasmic autoantibody-associated vasculitis and large-vessel vasculitis
por: Michailidou, Despina, et al.
Publicado: (2022)