Cargando…
Inhibition of overexpression of Giα proteins and nitroxidative stress contribute to sodium nitroprusside‐induced attenuation of high blood pressure in SHR
We earlier showed that vascular smooth muscle cells (VSMC) from spontaneously hypertensive rats (SHR) exhibit enhanced expression of Giα proteins which was attributed to the decreased levels of nitric oxide (NO), because elevation of the intracellular levels of NO by NO donors; sodium nitroprusside...
Autores principales: | Hossain, Ekhtear, Sarkar, Oli, Li, Yuan, Anand‐Srivastava, Madhu B. |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
John Wiley and Sons Inc.
2018
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5875540/ https://www.ncbi.nlm.nih.gov/pubmed/29595917 http://dx.doi.org/10.14814/phy2.13658 |
Ejemplares similares
-
Nitric oxide attenuates overexpression of Giα proteins in vascular smooth muscle cells from SHR: Role of ROS and ROS-mediated signaling
por: Sarkar, Oli, et al.
Publicado: (2017) -
Physiological Modeling of Hemodynamic Responses to Sodium Nitroprusside
por: Rinehart, Joseph, et al.
Publicado: (2023) -
Contribution of oxidative stress and growth factor receptor transactivation in natriuretic peptide receptor C-mediated attenuation of hyperproliferation of vascular smooth muscle cells from SHR
por: Rahali, Sofiane, et al.
Publicado: (2018) -
Sodium nitroprusside is not associated with metabolic acidosis during intraoperative infusion in children
por: Hammer, Gregory B, et al.
Publicado: (2013) -
Sodium nitroprusside in 2014: A clinical concepts review
por: Hottinger, Daniel G, et al.
Publicado: (2014)