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A Very Rare Case of Hypereosinophilic Syndrome Secondary to Natural Killer/T-Cell Lymphoma

Hypereosinophilic syndrome (HES) is a systemic disease characterized by an increased peripheral blood eosinophil count accompanied by systemic organ dysfunction. HES is classified into idiopathic HES, primary (neoplastic) HES (HES(N)), and secondary (reactive) HES (HES(R)). In this case report, a pa...

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Autores principales: Yamamoto, Takanori, Kamijo, Atsushi, Nakazawa, Tadao, Nakajima, Kei, Kirito, Keita, Komatsu, Norio, Masuyama, Keisuke
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Hindawi 2018
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5892269/
https://www.ncbi.nlm.nih.gov/pubmed/29785315
http://dx.doi.org/10.1155/2018/5965029
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author Yamamoto, Takanori
Kamijo, Atsushi
Nakazawa, Tadao
Nakajima, Kei
Kirito, Keita
Komatsu, Norio
Masuyama, Keisuke
author_facet Yamamoto, Takanori
Kamijo, Atsushi
Nakazawa, Tadao
Nakajima, Kei
Kirito, Keita
Komatsu, Norio
Masuyama, Keisuke
author_sort Yamamoto, Takanori
collection PubMed
description Hypereosinophilic syndrome (HES) is a systemic disease characterized by an increased peripheral blood eosinophil count accompanied by systemic organ dysfunction. HES is classified into idiopathic HES, primary (neoplastic) HES (HES(N)), and secondary (reactive) HES (HES(R)). In this case report, a patient who developed peripheral blood eosinophilia and granulation tissue in the pharynx and paranasal sinus, which was initially diagnosed as chronic eosinophilic leukemia (CEL), categorized as HES(N), but was eventually identified after the patient had died as natural killer/T-cell (NK/T) lymphoma, nasal type (ENKL), categorized as HES(R), is presented. ENKL-induced HES is very rare but must be considered.
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spelling pubmed-58922692018-05-21 A Very Rare Case of Hypereosinophilic Syndrome Secondary to Natural Killer/T-Cell Lymphoma Yamamoto, Takanori Kamijo, Atsushi Nakazawa, Tadao Nakajima, Kei Kirito, Keita Komatsu, Norio Masuyama, Keisuke Case Rep Otolaryngol Case Report Hypereosinophilic syndrome (HES) is a systemic disease characterized by an increased peripheral blood eosinophil count accompanied by systemic organ dysfunction. HES is classified into idiopathic HES, primary (neoplastic) HES (HES(N)), and secondary (reactive) HES (HES(R)). In this case report, a patient who developed peripheral blood eosinophilia and granulation tissue in the pharynx and paranasal sinus, which was initially diagnosed as chronic eosinophilic leukemia (CEL), categorized as HES(N), but was eventually identified after the patient had died as natural killer/T-cell (NK/T) lymphoma, nasal type (ENKL), categorized as HES(R), is presented. ENKL-induced HES is very rare but must be considered. Hindawi 2018-03-26 /pmc/articles/PMC5892269/ /pubmed/29785315 http://dx.doi.org/10.1155/2018/5965029 Text en Copyright © 2018 Takanori Yamamoto et al. http://creativecommons.org/licenses/by/4.0/ This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
spellingShingle Case Report
Yamamoto, Takanori
Kamijo, Atsushi
Nakazawa, Tadao
Nakajima, Kei
Kirito, Keita
Komatsu, Norio
Masuyama, Keisuke
A Very Rare Case of Hypereosinophilic Syndrome Secondary to Natural Killer/T-Cell Lymphoma
title A Very Rare Case of Hypereosinophilic Syndrome Secondary to Natural Killer/T-Cell Lymphoma
title_full A Very Rare Case of Hypereosinophilic Syndrome Secondary to Natural Killer/T-Cell Lymphoma
title_fullStr A Very Rare Case of Hypereosinophilic Syndrome Secondary to Natural Killer/T-Cell Lymphoma
title_full_unstemmed A Very Rare Case of Hypereosinophilic Syndrome Secondary to Natural Killer/T-Cell Lymphoma
title_short A Very Rare Case of Hypereosinophilic Syndrome Secondary to Natural Killer/T-Cell Lymphoma
title_sort very rare case of hypereosinophilic syndrome secondary to natural killer/t-cell lymphoma
topic Case Report
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5892269/
https://www.ncbi.nlm.nih.gov/pubmed/29785315
http://dx.doi.org/10.1155/2018/5965029
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