Cargando…

The Effects of PPAR Stimulation on Cardiac Metabolic Pathways in Barth Syndrome Mice

Aim: Tafazzin knockdown (TazKD) in mice is widely used to create an experimental model of Barth syndrome (BTHS) that exhibits dilated cardiomyopathy and impaired exercise capacity. Peroxisome proliferator-activated receptors (PPARs) are a group of nuclear receptor proteins that play essential roles...

Descripción completa

Detalles Bibliográficos
Autores principales:	Schafer, Caitlin, Moore, Vicky, Dasgupta, Nupur, Javadov, Sabzali, James, Jeanne F., Glukhov, Alexander I., Strauss, Arnold W., Khuchua, Zaza
Formato:	Online Artículo Texto
Lenguaje:	English
Publicado:	Frontiers Media S.A. 2018
Materias:	Pharmacology
Acceso en línea:	https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5904206/ https://www.ncbi.nlm.nih.gov/pubmed/29695963 http://dx.doi.org/10.3389/fphar.2018.00318

Ejemplares similares

Elucidating the Beneficial Role of PPAR Agonists in Cardiac Diseases
por: Khuchua, Zaza, et al.
Publicado: (2018)

The PPAR pan-agonist bezafibrate ameliorates cardiomyopathy in a mouse model of Barth syndrome
por: Huang, Yan, et al.
Publicado: (2017)

Cardiac Metabolic Pathways Affected in the Mouse Model of Barth Syndrome
por: Huang, Yan, et al.
Publicado: (2015)

Crosstalk between AMPK activation and angiotensin II-induced hypertrophy in cardiomyocytes: the role of mitochondria
por: Hernández, Jessica Soto, et al.
Publicado: (2014)

Diminished Exercise Capacity and Mitochondrial bc1 Complex Deficiency in Tafazzin-Knockdown Mice
por: Powers, Corey, et al.
Publicado: (2013)

The Role of PPARα in Metformin-Induced Attenuation of Mitochondrial Dysfunction in Acute Cardiac Ischemia/Reperfusion in Rats
por: Barreto-Torres, Giselle, et al.
Publicado: (2012)

Telomeres and Telomerase in Heart Ontogenesis, Aging and Regeneration
por: Nalobin, Denis, et al.
Publicado: (2020)

Barthes
por: Culler, Jonathan D.
Publicado: (1987)

Introducing Barthes /
por: Thody, Philip Malcolm Waller, 1928-
Publicado: (1999)

Barth syndrome
por: JEFFERIES, JOHN L
Publicado: (2013)

Barth syndrome
por: Clarke, Sarah LN, et al.
Publicado: (2013)

Possible Role of Interaction between PPARα and Cyclophilin D in Cardioprotection of AMPK against In Vivo Ischemia-Reperfusion in Rats
por: Barreto-Torres, Giselle, et al.
Publicado: (2016)

Cardiac‐specific succinate dehydrogenase deficiency in Barth syndrome
por: Dudek, Jan, et al.
Publicado: (2015)

Barthes para principiantes /
por: Thody, Philip Malcolm Waller, 1928-
Publicado: (1997)

High Sensitivity of SIRT3 Deficient Hearts to Ischemia-Reperfusion Is Associated with Mitochondrial Abnormalities
por: Parodi-Rullán, Rebecca M., et al.
Publicado: (2017)

Corrigendum: High Sensitivity of SIRT3 Deficient Hearts to Ischemia-Reperfusion Is Associated with Mitochondrial Abnormalities
por: Parodi-Rullán, Rebecca M., et al.
Publicado: (2017)

The calcium-ROS-pH triangle and mitochondrial permeability transition: challenges to mimic cardiac ischemia-reperfusion
por: Javadov, Sabzali
Publicado: (2015)

Oeuvres choisies de Barthe.
por: Barthe, Nicolas-Thomas (1736-1785)
Publicado: (1811)

Roland Barthes, 1915-1980 /
por: Calvet, Louis Jean, 1942-
Publicado: (1992)

Barth syndrome: mechanisms and management
por: Finsterer, Josef
Publicado: (2019)

Barth Syndrome Cardiomyopathy: An Update
por: Pang, Jing, et al.
Publicado: (2022)

Quality of life in Barth syndrome
por: Kim, Alexander Y., et al.
Publicado: (2022)

The Natural PPAR Agonist Linoleic Acid Stimulated Insulin Release in the Rat Pancreas
por: LAI, Min-Chuan, et al.
Publicado: (2013)

Elucidating the contribution of ETC complexes I and II to the respirasome formation in cardiac mitochondria
por: Jang, Sehwan, et al.
Publicado: (2018)

Activation of the integrated stress response rewires cardiac metabolism in Barth syndrome
por: Kutschka, Ilona, et al.
Publicado: (2023)

Barthes en ensayo : introducción al texto reflexionante /
por: Bensmaïa, Réda
Publicado: (2017)

Roland Barthes : de las críticas de interpretación al análisis textual /
por: Sirvent Ramos, Angeles
Publicado: (1989)

Dapagliflozin Mediates Plin5/PPARα Signaling Axis to Attenuate Cardiac Hypertrophy
por: Yu, Jing, et al.
Publicado: (2021)

Stimulating myocardial pyruvate dehydrogenase activity fails to alleviate cardiac abnormalities in a mouse model of human Barth syndrome
por: Greenwell, Amanda A., et al.
Publicado: (2022)

Las muertes de Roland Barthes
por: Derrida, Jacques

Obituary Notice of Dr Barth of Paris
Publicado: (1878)

Pioglitazone, a PPAR-γ Activator, Stimulates BK(Ca) but Suppresses IK(M) in Hippocampal Neurons
por: Chen, Tsang-Shan, et al.
Publicado: (2018)

Commentary: Dapagliflozin Mediates Plin5/PPARα Signaling Axis to Attenuate Cardiac Hypertrophy
por: Liu, Zengying, et al.
Publicado: (2022)

New clinical and molecular insights on Barth syndrome
por: Ferri, Lorenzo, et al.
Publicado: (2013)

Assessing olfactory functions in patients with Barth syndrome
por: Dibattista, Michele, et al.
Publicado: (2017)

Barth syndrome cardiomyopathy: targeting the mitochondria with elamipretide
por: Sabbah, Hani N.
Publicado: (2020)

Myocardial disturbances of intermediary metabolism in Barth syndrome
por: Greenwell, Amanda A., et al.
Publicado: (2022)

Impaired cardiac and skeletal muscle bioenergetics in children, adolescents, and young adults with Barth syndrome
por: Bashir, Adil, et al.
Publicado: (2017)

Barth Syndrome: Exploring Cardiac Metabolism with Induced Pluripotent Stem Cell-Derived Cardiomyocytes
por: Fatica, Erica M., et al.
Publicado: (2019)

Extended recovery of cardiac function after severe infantile cardiomyopathy presentation of Barth syndrome
por: Yester, Jessie, et al.
Publicado: (2021)

Cannot write session to /tmp/vufind_sessions/sess_eivp2dcblr6rum7a1c4vq0vnvm