TAK1 mediates excessive autophagy via p38 and ERK in cisplatin‐induced acute kidney injury

The ability of cisplatin (cis‐diamminedichloroplatinum II) toxicity to induce acute kidney injury (AKI) has attracted people's attention and concern for a long time, but its molecular mechanisms are still widely unknown. We found that the expression of transforming growth factor‐β (TGF‐β)‐activ...

Descripción completa

Detalles Bibliográficos
Autores principales: Zhou, Jun, Fan, Youling, Zhong, Jiying, Huang, Zhenxing, Huang, Teng, Lin, Sen, Chen, Hongtao
Formato: Online Artículo Texto
Lenguaje:English
Publicado: John Wiley and Sons Inc. 2018
Materias:
Original Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5908118/
https://www.ncbi.nlm.nih.gov/pubmed/29504713
http://dx.doi.org/10.1111/jcmm.13585

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5908118/
https://www.ncbi.nlm.nih.gov/pubmed/29504713
http://dx.doi.org/10.1111/jcmm.13585

Ejemplares similares