Inhibition of Epac2 Attenuates Neural Cell Apoptosis and Improves Neurological Deficits in a Rat Model of Traumatic Brain Injury

Traumatic brain injury (TBI) is a major cause of mortality and disability worldwide. TBI-induced neuronal apoptosis is one of the main contributors to the secondary injury process. The aim of this study is to investigate the involvement of Exchange protein directly activated by cAMP 2 (Epac2) on TBI...

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Detalles Bibliográficos
Autores principales: Zhang, Ling, Zhang, Li, Liu, Huixiang, Jiang, Feng, Wang, Huanjing, Li, Di, Gao, Rong
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Frontiers Media S.A. 2018
Materias:
Neuroscience
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5924794/
https://www.ncbi.nlm.nih.gov/pubmed/29740274
http://dx.doi.org/10.3389/fnins.2018.00263

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5924794/
https://www.ncbi.nlm.nih.gov/pubmed/29740274
http://dx.doi.org/10.3389/fnins.2018.00263

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