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How to tackle congestion in acute heart failure

Acute heart failure is a common complication of chronic heart failure and is associated with a high risk for subsequent mortality and morbidity. In 90% of case acute heart failure is the resultant of congestion, a manifestation of fluid build-up due to increased filling pressures. As residual conges...

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Detalles Bibliográficos
Autores principales: Martens, Pieter, Mullens, Wilfried
Formato: Online Artículo Texto
Lenguaje:English
Publicado: The Korean Association of Internal Medicine 2018
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5943665/
https://www.ncbi.nlm.nih.gov/pubmed/29627971
http://dx.doi.org/10.3904/kjim.2017.355
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author Martens, Pieter
Mullens, Wilfried
author_facet Martens, Pieter
Mullens, Wilfried
author_sort Martens, Pieter
collection PubMed
description Acute heart failure is a common complication of chronic heart failure and is associated with a high risk for subsequent mortality and morbidity. In 90% of case acute heart failure is the resultant of congestion, a manifestation of fluid build-up due to increased filling pressures. As residual congestion at discharge following an acute heart failure episodes is one of the strongest predictors of poor outcome, the goal of therapy should be to resolve congestion completely. Important to comprehend is that increased cardiovascular filling pressures are not solely the resultant of intravascular volume excess but can also be induced by a decreased venous capacitance. This review article focusses on the pathophysiology, diagnoses, and treatment of congestion in acute heart failure. A clear distinction is made between states of volume overload (intravascular volume excess) or volume redistribution (decreased venous capacitance) contributing to congestion in acute heart failure.
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spelling pubmed-59436652018-05-16 How to tackle congestion in acute heart failure Martens, Pieter Mullens, Wilfried Korean J Intern Med Review Acute heart failure is a common complication of chronic heart failure and is associated with a high risk for subsequent mortality and morbidity. In 90% of case acute heart failure is the resultant of congestion, a manifestation of fluid build-up due to increased filling pressures. As residual congestion at discharge following an acute heart failure episodes is one of the strongest predictors of poor outcome, the goal of therapy should be to resolve congestion completely. Important to comprehend is that increased cardiovascular filling pressures are not solely the resultant of intravascular volume excess but can also be induced by a decreased venous capacitance. This review article focusses on the pathophysiology, diagnoses, and treatment of congestion in acute heart failure. A clear distinction is made between states of volume overload (intravascular volume excess) or volume redistribution (decreased venous capacitance) contributing to congestion in acute heart failure. The Korean Association of Internal Medicine 2018-05 2018-04-11 /pmc/articles/PMC5943665/ /pubmed/29627971 http://dx.doi.org/10.3904/kjim.2017.355 Text en Copyright © 2018 The Korean Association of Internal Medicine This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/4.0/) which permits unrestricted noncommercial use, distribution, and reproduction in any medium, provided the original work is properly cited.
spellingShingle Review
Martens, Pieter
Mullens, Wilfried
How to tackle congestion in acute heart failure
title How to tackle congestion in acute heart failure
title_full How to tackle congestion in acute heart failure
title_fullStr How to tackle congestion in acute heart failure
title_full_unstemmed How to tackle congestion in acute heart failure
title_short How to tackle congestion in acute heart failure
title_sort how to tackle congestion in acute heart failure
topic Review
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5943665/
https://www.ncbi.nlm.nih.gov/pubmed/29627971
http://dx.doi.org/10.3904/kjim.2017.355
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