Cargando…
Special AT‐rich sequence binding protein 1 is required for maintenance of T cell receptor responsiveness and development of experimental autoimmune encephalomyelitis
The genome organizer special AT‐rich sequence binding protein 1 (SATB1) regulates specific functions through chromatin remodeling in T helper cells. It was recently reported by our team that T cells from SATB1 conditional knockout (SATB1cKO) mice, in which the Satb1 gene is deleted from hematopoieti...
Autores principales: | Akiba, Yasushi, Kuwabara, Taku, Mukozu, Takanori, Mikami, Tetuo, Kondo, Motonari |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
John Wiley and Sons Inc.
2018
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5947310/ https://www.ncbi.nlm.nih.gov/pubmed/29388727 http://dx.doi.org/10.1111/1348-0421.12579 |
Ejemplares similares
-
Regulation of T-Cell Signaling by Post-Translational Modifications in Autoimmune Disease
por: Kuwabara, Taku, et al.
Publicado: (2018) -
The Role of IL-17 and Related Cytokines in Inflammatory Autoimmune Diseases
por: Kuwabara, Taku, et al.
Publicado: (2017) -
Increased Indoleamine 2,3-Dioxygenase Levels at the Onset of Sjögren’s Syndrome in SATB1-Conditional Knockout Mice
por: Tanaka, Yuriko, et al.
Publicado: (2021) -
Regulation of Experimental Autoimmune Encephalomyelitis by Natural Killer (NK) Cells
por: Zhang, Ben-ning, et al.
Publicado: (1997) -
Identification of an Essential Cytoplasmic Region of Interleukin-7 Receptor α Subunit in B-Cell Development
por: Kasai, Hirotake, et al.
Publicado: (2018)