Blockage of store-operated Ca(2+) entry antagonizes Epstein–Barr virus-promoted angiogenesis by inhibiting Ca(2+) signaling-regulated VEGF production in nasopharyngeal carcinoma

BACKGROUND: Epstein–Barr virus (EBV) actively contributes to the pathological process of nasopharyngeal carcinoma (NPC) by enabling NPC cells to acquire various capacities required for their malignant biological actions. Our earlier works demonstrated that EBV-encoded latent membrane protein 1 (LMP1...

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Detalles Bibliográficos
Autores principales: Ye, Jiaxiang, Huang, Junqi, He, Qian, Zhao, Weilin, Zhou, Xiaoying, Zhang, Zhe, Li, Yongqiang, Wei, Jiazhang, Zhang, Jinyan
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Dove Medical Press 2018
Materias:
Original Research
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5955047/
https://www.ncbi.nlm.nih.gov/pubmed/29785139
http://dx.doi.org/10.2147/CMAR.S159441

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5955047/
https://www.ncbi.nlm.nih.gov/pubmed/29785139
http://dx.doi.org/10.2147/CMAR.S159441

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