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Deficiency of NOD1 Improves the β-Adrenergic Modulation of Ca(2+) Handling in a Mouse Model of Heart Failure
Heart failure (HF) is a complex syndrome characterized by cardiac dysfunction, Ca(2+) mishandling, and chronic activation of the innate immune system. Reduced cardiac output in HF leads to compensatory mechanisms via activation of the adrenergic nervous system. In turn, chronic adrenergic overstimul...
Autores principales: | , , , , , , , , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Frontiers Media S.A.
2018
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6010671/ https://www.ncbi.nlm.nih.gov/pubmed/29962957 http://dx.doi.org/10.3389/fphys.2018.00702 |