Cargando…
Developmental changes of GABA immunoreactivity in cortico-thalamic networks of an absence seizure model
Absence seizures (ASs) are associated with abnormalities in gamma-aminobutyric acid (GABA) neurotransmission in the thalamus and the cortex. In the present study, we used light microscopy GABA immunocytochemistry to quantify the GABA-immunoreactive (GABA-IR) neurons and neuropil in the thalamic vent...
Autores principales: | Bombardi, Cristiano, Venzi, Marcello, Crunelli, Vincenzo, Di Giovanni, Giuseppe |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Pergamon Press
2018
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6018618/ https://www.ncbi.nlm.nih.gov/pubmed/29471054 http://dx.doi.org/10.1016/j.neuropharm.2018.01.047 |
Ejemplares similares
-
Role for serotonin2A (5-HT2A) and 2C (5-HT2C) receptors in experimental absence seizures
por: Venzi, Marcello, et al.
Publicado: (2016) -
A Critical Evaluation of the Gamma‐Hydroxybutyrate (GHB) Model of Absence Seizures
por: Venzi, Marcello, et al.
Publicado: (2014) -
Cortical drive and thalamic feed-forward inhibition control thalamic output synchrony during absence seizures
por: McCafferty, Cian, et al.
Publicado: (2018) -
Aberrant GABA(A) Receptor-Mediated Inhibition in Cortico-Thalamic Networks of Succinic Semialdehyde Dehydrogenase Deficient Mice
por: Errington, Adam C., et al.
Publicado: (2011) -
Transition to absence seizures and the role of GABA(A) receptors
por: Crunelli, Vincenzo, et al.
Publicado: (2011)