Cargando…
P31–43, an undigested gliadin peptide, mimics and enhances the innate immune response to viruses and interferes with endocytic trafficking: a role in celiac disease
Celiac disease (CD) is an autoimmune disease characterized by inflammation of the intestinal mucosa due to an immune response to wheat gliadins. Some gliadin peptides are resistant to intestinal digestion (e.g., A-gliadin P31–43) and induce a stress/innate immune response, but the reason why they ar...
Autores principales: | Nanayakkara, Merlin, Lania, Giuliana, Maglio, Mariantonia, Auricchio, Renata, De Musis, Cristiana, Discepolo, Valentina, Miele, Erasmo, Jabri, Bana, Troncone, Riccardo, Auricchio, Salvatore, Barone, Maria Vittoria |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Nature Publishing Group UK
2018
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6050301/ https://www.ncbi.nlm.nih.gov/pubmed/30018339 http://dx.doi.org/10.1038/s41598-018-28830-y |
Ejemplares similares
-
Constitutive alterations in vesicular trafficking increase the sensitivity of cells from celiac disease patients to gliadin
por: Lania, Giuliana, et al.
Publicado: (2019) -
Gliadin-Mediated Proliferation and Innate Immune Activation in Celiac Disease Are Due to Alterations in Vesicular Trafficking
por: Barone, M. Vittoria, et al.
Publicado: (2011) -
Author Correction: Constitutive alterations in vesicular trafficking increase the sensitivity of cells from celiac disease patients to gliadin
por: Lania, Giuliana, et al.
Publicado: (2020) -
Gliadin Peptide P31-43 Localises to Endocytic Vesicles and Interferes with Their Maturation
por: Barone, Maria Vittoria, et al.
Publicado: (2010) -
Enterocyte Proliferation and Signaling Are Constitutively Altered in Celiac Disease
por: Nanayakkara, Merlin, et al.
Publicado: (2013)